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Fig. 1 | Gut Pathogens

Fig. 1

From: Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression

Fig. 1

Phase contrast microscopy and quantification of elongated cells of a gastric cell line and three oral epithelial cell lines infected with H. pylori. Adherent cells of gastric AGS (a) and oral CAL-27 (b), HN (c) and BHY (d) cells were co-incubated with H. pylori wild-type strain Gam94-24 for 6 h. Panels to the left show control cells, and to the right the corresponding infected cells are shown. Cell elongation is only visible upon infection of AGS cells. Quantitative cell elongation (grey bars) and bacterial cell binding (black bars) results from triplicate experiments are shown for AGS (e), CAL-27 (f), HN (g) and BHY (h) cells following infection with the indicated seven H. pylori wild-type strains and a ∆cagY mutant of CUZ20 for 6 h (ns, not significant)

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