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Fig. 2 | Gut Pathogens

Fig. 2

From: Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression

Fig. 2

CagA internalization in host cells upon H. pylori infection visualized by Western blots. ad Show Western blots stained with α-PY-99 antibodies for detection of internalized, phosphorylated CagAPY on top (CagAPY position indicated by an arrow), non-phosphorylated CagA detected by α-CagA antibodies as a bacterial loading control in the middle, and the β-actin protein loading control at the bottom, followed by the relative fraction of CagAPY as determined by densitometry. a Shows AGS cells infected with the seven wild-type H. pylori strains plus the ∆cagY mutant for 6 h, and bd) show those results for HN, CAL-27 and BHY cells. The asterisks in the α-PY-99-stained blots indicate the position of a ~ 125 kDa host cell protein

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