| • Direct protection of the intestinal barrier |
| • Influence on local and systemic antioxidant status, reduction in lipid peroxidation |
| • Direct, microbial-produced neurochemical production – e.g. GABA |
| • Indirect influence on neurotransmitter/neuropeptide production |
| • Prevention of stress-induced alterations to overall intestinal microbiota |
| • Direct activation of neural pathways between gut and brain |
| • Limitation of inflammatory cytokine production |
| • Modulation of neurotrophic chemicals including brain-derived neurotrophic factor |
| • Limitation of carbohydrate malabsorption |
| • Improvement of nutritional status – e.g. omega-3 fatty acids, minerals, phytochemicals |
| • Limitation of small intestinal bacterial overgrowth |
| • Reduction of amine/uremic toxin burden |
| • Limitation of gastric/intestinal pathogens (e.g. Helicobacter pylori) |
| • Analgesic properties |